Before I begin, this will probably be the last blog post. I will probably write a reflection of the project in my DP (students.hightechhigh.org/~jrubio). Now, about Cipro.
"...inside E. Coli bacteria and anthrax bacteria is an enzyme, called topoisomerase II, that helps the cell to wind DNA into a compact structure and then unwind it when needed. Cipro blocks topoisomerase II and prevents it from doing its job. A bacterial cell that has Cipro in it can no longer uncoil its DNA in order to create enzymes or reproduce. The bacteria containing Cipro eventually die." This is straight from How Stuff Works. (http://health.howstuffworks.com/cipro3.htm)
This pretty much covers it. I'll try to simplify it a bit. In simpler terms, Cipro prevents bacteria from reproducing. The remaining bacteria, eventually dies off. Cipro would be effective against Cholera in the same manner, by preventing Cholera from multiplying the bacteria will die off. This will make Cholera not last as long because there won't be any new bacteria, and it won't be as severe, because there's not as many Cholera bacteria in the system. This is why it's important to take Cipro in the early stage, and not stop taking it for awhile, or resistance will develop.
On a last note, it looks like we'll finish in time, and check back my DP for updates, and work samples.
1 comment:
All of which is true, the only thing to do now is to get these around the world.
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